Hypoxic Ischemic Encephalopathy (H.I.E.)Birth injury lawyers skilled in preventable hypoxia and ischemia cases
Newborn up to 20 years old. Free birth injury case evaluation.
Hypoxic ischemic encephalopathy is usually caused by a lack of oxygen to the an infant’s brain at some during a pregnancy. The results of a lack of oxygen for any period of time could be a permanent injury to the infant. In some cases, the circumstances that led to the lack of oxygen could have been avoided. In this case, the child’s injuries were preventable and this could bring about the decision to file a claim. If you believe this to be the case, and the child is newborn or born less than 20 years ago, contact us for a free consultation to the get the answers you deserve.
At Latona Law, we are experienced birth injury attorneys, specialized in cases where negligence led to hypoxia, ischemia or any condition due to prolonged lack of oxygen to the infant. We have helped many families in the Wilkes-Barre/Scranton area, get justice for their child’s hypoxic ischemic encephalopathy, or other long-term condition.
Hypoxia/Ischemia Injuries: Things to consider
- Injuries suffered by a baby can take place before, during, or after birth.
- Birth injuries happen during difficult deliveries, emergency c-sections or other out of the ordinary events, such as placental abruption.
- Consequently, oxygen to the infant’s brain may be restricted, ultimately causing the injury.
- If there were medical errors, your family may be able to recover compensation for damages. This includes past and current medical expenses, in addition to any future care required.
Contact us below or call (570)825-9000 for a FREE hypoxic ischemia case evaluation:
A 2008 study of 386 infant patients who suffered any form of hypoxia ischemia found significant affects and disabilities throughout life, including 14.2% that qualified for a cerebral palsy diagnosis. Other results include seizures, neurological deficits, and possibly death.
Hypoxic Brain Damage Symptoms
Symptoms can appear mild, such as dizziness and lack of concentration, although long-term symptoms can include more serious complications like damage to the cerebral cortex, the hippocampus, and occipital lobe. This can cause issues with brain development, including learning reading and linguistic skills, as well as other learning disabilities.
- Mild hypoxic-ischemic encephalopathy
- Muscle tone may be slightly increased and deep tendon reflexes may be brisk during the first few days
- Transient behavioral abnormalities, such as poor feeding, irritability, or excessive crying or sleepiness (typically in an alternating pattern)
- Typically resolves in 24 hours
Moderately severe hypoxic-ischemic encephalopathy
- The infant is lethargic, with significant hypotonia and diminished deep tendon reflexes
- The grasping, Moro, and sucking reflexes may be sluggish or absent
- The infant may experience occasional periods of apnea
- Seizures typically occur early within the first 24 hours after birth
- Full recovery within 1-2 weeks is possible and is associated with a better long-term outcome
Severe hypoxic-ischemic encephalopathy
- Seizures can be delayed and severe and may be initially resistant to conventional treatments
- The seizures are usually generalized, and their frequency may increase during the 24-48 hours after onset, correlating with the phase of reperfusion injury
- As the injury progresses, seizures subside and the electroencephalogram becomes isoelectric or shows a burst suppression pattern. At that time, wakefulness may deteriorate further, and the fontanelle may bulge, suggesting increasing cerebral edema.
- Stupor or coma is typical; the infant may not respond to any physical stimulus except the most noxious
- Breathing may be irregular, and the infant often requires ventilator support
- Generalized hypotonia and depressed deep tendon reflexes are common
- Neonatal reflexes (sucking, swallowing, grasping, Moro) are absent
- Disturbances of ocular motion, such as a skewed deviation of the eyes, nystagmus, bobbing, and loss of “doll’s eye (ie, conjugate) movements may be revealed by cranial nerve examination
- Pupils may be dilated, fixed, or poorly reactive to light
- Irregularities of heart rate and blood pressure are common during the period of reperfusion injury, as is death from cardiorespiratory failure
- An initial period of well-being or mild hypoxic-ischemic encephalopathy may be followed by sudden deterioration, suggesting ongoing brain cell dysfunction, injury, and death; during this period, seizure intensity may increase
- Causes of HIE include the following:
- Placental insufficiencies, placental abrution, placental blood clots
- Maternal diabetes
- Maternal cardiovascular collapse
- Maternal blood clotting disorders
- Maternal low blood pressure
- Fetal maternal hemorrhage
- Meconium aspiration, aspiration causing asphyxia event
- Umbilical cord accidents/issures, cord prolapse, true umbilical knots, cord compression
- Uterine rupture
- Prolonged late stages of labor, failure to progress, trauma during deliver
- Abnormal fetal position, shoulder dystocia
- Infections, viral infections, bacterial or viral infections such as meningitis or sepsis
- Brain skull trauma
Signs and Signals of H.I.E.
- There are many signs to look for in an infant with Hypoxic Ischemic Encephalopathy:
- Tense and react more to stimulation than most newborns
- Floppy and hardly react to stimulation
- Visually inactive (no eye contact)
- Move abnormally or even have seizures
- Having problems feeding because their suck is too weak and the muscles in their mouth and throat are not working properly
- Display a very weak cry
- Meconium-stained amniotic fluid
- Low heart rate
- Poor muscle tone
- Weak breathing or no breathing at all
- Bluish or pale skin color
- Excessive acid in the blood